By H. E. Kaiser (auth.), Arthur S. Levine (eds.)
This quantity experiences our present wisdom touching on can numerous chapters talk about the contributions of genetic asp cer progress and development because it pertains to the etiology of ects, metabolism, endocrine-related facets and food to human melanoma. As emphasised in Volumes I-V of this sequence, melanoma development. in addition, our present wisdom con neoplastic ailments are multistep maladies. there are lots of cerning urbanization elements, radiation, therapy-induced factors for the looks of neoplastic ailments. prior neoplasms, environmentally brought about neoplasms (e. g. , chapters within the sequence have reviewed molecular and mobile mesotheliomas caused via asbestos) and malignant neo elements of tumor initiation, promoting and development to plasms in organ transplant recipients are summarized. the invasive and metastatic phenotype. Contributions to the The effect of AIDS on neoplasm improvement is re initiation and development of neoplastic ailments are made via considered from an epidemiologic standpoint that explores mul average positive aspects of our environment and through its contaminants tiple features of immunity, infectious disorder, sexual habit and via dietary components. Neoplastic ailments convey a dis and blood transfusion. different chapters examine the in tinct courting to numerous environmental stimuli and fluence of the host immune reaction in oncogenesis and the to illnesses of a non-neoplastic nature. for instance, familial courting among atherosclerotic plaques and tumors.
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Extra resources for Etiology of Cancer in Man
Br J Hael11atoI29:575, 1975 123. Trage KH, Mayre WR: Familial islet-cell adenomatosis. Lancet 2:426, 1977 124. Tyzzer EE: A study of heredity in relation to the development of tumors in mice. J Med Res 17:199, 1907 125. Van Den Berghe H, Parloir C, Gosseye S, Englebieme V, Cornu G, Sokal G: Variant translocation in Burkitt lymphoma. Cancer Genet Cytogenet 1:9, 1979 126. Weber W, Ott J, Gencik A, Muller HJ: Familial cancer: genetically determined? Anticancer Research 3: 133, 1982 127. Wennstrom J, Pierce ER, McKusik VA: Hereditary benign and malignant lesions of the large bowel.
GG, Dolby ThW, Koprowski H: Chromosomal location of the genes for human immunoglobulin heavy chains. Proc NaIl Acad Sci USA 76:3416, 1979 Dalla-Favera R, Bregni M, Erikson J, Patterson D, Gallo RC, Croce CM: Human c-myc onc gene is located on the region of chromosome 8 that is translocated in Burkitt lymphoma cells. Proc Natl Acad Sci USA 79:7824, 1982 Delmas-Marsalet Y, Hors J, Colombani J, Dausett J: Study of HL-A genotypes in a case of familial chronic lymphocytic leukemia (CLL). Tissue Antigens 4:441, 1974 Donahue WL, Thompson MW: Familial Iymphohistiocytosis.
14 B ,, ... 14 B ,, • .. m Figure 6. Normal chromosome 8 and 14 and marker chromosomes - marker 8 and marker 14 from 6 cells of the Burkitt lymphoma cell lines. Daudi and Jijoye, presented with increasing contraction in progressive mitotic stages from prosomes (prophase-prometaphase chromosomes) to mesomes (metaphase chromosomes). T. et al. Epstein-Barr Virus as an Etiological Agent in the Pathogenesis of Lymphoproliferative and Aproliferative Diseases in Immune Deficient Patients. W. A. Epstein (eds).
Etiology of Cancer in Man by H. E. Kaiser (auth.), Arthur S. Levine (eds.)