Ames, David; Burns, Alistair S.; O'Brien, John's Dementia PDF

By Ames, David; Burns, Alistair S.; O'Brien, John

ISBN-10: 0340987278

ISBN-13: 9780340987278

ISBN-10: 1444128744

ISBN-13: 9781444128741

ISBN-10: 1461568056

ISBN-13: 9781461568056

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327–8). Years later, Jackson (1875) reviewed the problem: ‘softening y as a category for a rude clinical grouping was to be deprecated’ (p. 335); he nonetheless followed DurandFardel’s classification and suggested that, after stroke, mental symptoms might be immediate or occurring after a few hours or months; he recognized that major cognitive failure may ensue, and saw this as an instance of ‘dissolution’: emotional symptoms being release phenomena (for an analysis of this concept see Berrios, 1991).

115). On this definition, it is difficult, on the basis of their intrinsic features, to distinguish illusion of memory from confabulation. Indeed, symptom-naming is determined by whether schizophrenia or ‘organic disorder’ is the associated disease. g. memory hallucinations which fill in memory gaps which at first appear at a (usually external) given moment and mostly adapt themselves to such an occasion’ (p. 117). Kleist (1960) described patients with ‘progressive confabulosis’ as ‘cheerful, expansive, and with little in the way of thought or speech disorder’ (p.

Kraepelin (1910) lumped presbyophrenia together with the senile and presenile insanities, and (as compared with Korsakoff ’s patients) believed presbyophrenic patients to be older, free from polyneuritis and history of alcoholism, and showing hyperactivity and elevated mood. Ziehen (1911) wrote that ‘their marked memory impairment contrasts with the relative sparing of thinking’. Fischer (1912) suggested that disseminated cerebral lesions were the essential anatomical substratum of presbyophrenia.

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Dementia by Ames, David; Burns, Alistair S.; O'Brien, John


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